Journal of Ocular Biology

Case Report

Treating Acquired Optic Disc Pit Maculopathy with an Ocular Hypotensive Agent

Alex L Song, Mark P. Ghassibi and Eric Shrier*

Department of Ophthalmology, SUNY Downstate Health Sciences University, Brooklyn, NY, USA
*Address for Correspondence: Eric Shrier DO, Department of Ophthalmology, SUNY Downstate Health Sciences University, 450 Clarkson Ave., Room B7-302, MSC 58 Brooklyn, NY 11203, USA Tel: 718-270-1714 Email: eric.shrier@downstate.edu
Submission: 27-June-2023 Accepted: 21-July-2023 Published: 24-July-2023
Copyright: © 2023 Song AL, et al. This is an open-access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

Abstract

Purpose: To describe a case of glaucomatous optic disc pit maculopathy that resolved after bimatoprost treatment. Observations: A 63-year-old African American female with a history of type 2 diabetes mellitus presented to the clinic after 3 months of blurry vision in the right eye and was found to have an asymmetric cup-to-disc ratio, and right intraretinal and subretinal fluid without leakage on fluorescein angiography concerning for optic disc pit maculopathy. A trial of intravitreal anti-vascular endothelial growth factor (VEGF) injection failed to resolve the fluid. Optical coherence tomography of the ganglion cell complex showed thinning of the ganglion cell complex concerning for previously undiagnosed glaucoma, and the patient was started on bimatoprost. The previously noted fluid resolved, and the patient has been quiescent for 4 years on ocular hypotensive medication.
Conclusion: In untreated or poorly controlled glaucoma, progressive cupping of the optic nerve head may lead to the development of an acquired optic disc pit, which can produce maculopathy. This maculopathy can improve under ocular hypotensive treatment.